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Toll‐like receptors (TLRs) and P2 receptors are key regulators of innate immunity. During infection, pathogen‐associated molecular patterns (PAMPs) activate TLRs, whereas extracellular nucleotides engage P2 receptors and influence inflammatory responses. Here, we show that polyinosinic:polycytidylic acid (poly[I:C])‐ac...
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