Author

Anil K. Rustgi

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Oct 2026

Aberrant Accumulation of Cyclin D1 Rewires Glutamine Metabolism and Generates a Metabolic Vulnerability in Squamous Cell Carcinoma.

Excessive cyclin D1 accumulation is a frequent oncogenic event in head and neck squamous cell carcinoma (HNSCC) and esophageal squamous cell carcinoma (ESCC). Although classically associated with enhanced proliferation and genomic instability, we revealed that cyclin D1 dysregulation induces metabolic rewiring that lea...

Shu-Yuan Zhang, Polly J. Phillips-Mason, B. Mucha et al. · Cancer Research · 0 citations

Open access Aug 2026

Stress granules confer an early vulnerability in KRAS-driven pancreatic tumorigenesis.

It is shown that stress granules (SGs) formation is a key adaptive mechanism enabling pancreatic cells to tolerate oncogenic KRAS signaling, and SGs inhibition does not affect pancreatic damage during chronic pancreatitis, supporting its safety for selectively targeting KRAS-mutant cells.

Matías Estarás, Emma Cosialls, Gareth Pollin et al. · Cell Reports · 0 citations

Open access Jul 2026

Molecular dynamics driving phenotypic divergence among KRAS mutants in pancreatic tumorigenesis.

To define mutation-specific lineage reversion and tumor initiation, Ptf1a-tdTomato mice and multiple KRAS mutants are implemented across several genetic, pharmacologic, and inflammatory perturbations in vivo to deciphering mutation-specific oncogenic trajectories and directing the implementation of KRAS-directed therap...

A. Grimont, David Falvo, W. Sisso et al. · Developmental Cell · 1 citation