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C. Focaccetti

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Open access Sep 2026

Telomeric Replication Stress Promotes ALT-Associated Hallmarks in ATRX- and p53-Deficient Cells.

BACKGROUND Most human tumors maintain telomeres through telomerase activation; however, a subset relies on the telomerase-independent mechanism known as Alternative Lengthening of Telomeres (ALT). ALT-positive tumors are frequently characterized by inactivating alterations in ATRX (Alpha Thalassemia/Mental Retardation...

Roberta Amato, Emanuela Micheli, C. Focaccetti et al. · 0 citations

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