Open access
Aug 2026
Complement activation linked to type II interferon signaling in Still disease.
OBJECTIVE Still disease (SD) is an autoinflammatory syndrome characterized by innate immune dysregulation. While complement can drive inflammation, its involvement in SD remains to be defined. Thus, we aimed to assess complement activation in SD. METHODS Complement was assessed using transcriptomic, proteomic, and in...
F. Huijsmans, Tabea Thalheim, A. Bodelón et al.
· Arthritis & Rheumatology · 0 citations