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Sponsored AI Networking Cookbook: Practical recipes for AI-assisted network automation and development AI Networking Cookbook: Practical recipes for AI-assisted network automation and development

Author

E. Gallardo

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4 papers indexed here

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Open access Nov 2026

Disease Control in Generalized Myasthenia Gravis: Consensus Definition, Evolution, and Patient Characterization.

BACKGROUND AND OBJECTIVES Generalized myasthenia gravis (gMG) is a chronic fluctuating neuromuscular disease. Some patients keep experiencing symptoms or adverse events (AEs) despite tailored therapeutic schemes. The aim of this study was to characterize adequately controlled gMG and describe patients' evolution and ou...

D. Reyes-Leiva, Á. Carbayo, A. Vesperinas et al. · 0 citations
Open access Sep 2026

Spatial transcriptomics reveals early and progressive molecular changes in late-onset Pompe disease muscle

Late-onset Pompe disease (LOPD) is caused by acid alpha-glucosidase deficiency, leading to lysosomal glycogen accumulation and progressive muscle pathology. Muscle biopsies show marked fibre heterogeneity, including non-vacuolated and vacuolated fibres. This study aimed to define transcriptomic alterations across the...

Alexandra Monceau, E. Gallardo, A. Hernández-Laín et al. · 0 citations
Case report Open access Aug 2026

Case Report: Pediatric immune-mediated necrotizing myopathies mimicking inherited muscle disorders: clinical, paraclinical, and genetic insights from two cases

Objectives Immune-mediated necrotizing myopathies (IMNMs) are a group of rare acquired myopathies. We describe two pediatric cases presenting with early-onset proximal and axial muscle weakness, rhabdomyolysis, and progressive evolution mimicking limb-girdle muscular dystrophy. Methods This retrospective case series is...

Gianmarco Severa, María Camila Cortés-Rojas, M. Gómez-García de la Banda et al. · 0 citations
Open access Aug 2026

NE06 Molecular mechanisms of persistent endocrine myopathy: beyond biochemical remission

Findings support the existence of a persistent molecular footprint in skeletal muscle after biochemical remission and suggest a shared epigenetic memory of prior GH or cortisol excess may contribute to long-term muscle dysfunction and represents a potential target for therapeutic intervention.

J. Gil, L. M. Duguech, R. Díaz et al. · 0 citations

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