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It is demonstrated that sample sizes in the hundreds are required for robust associations with psychiatric diagnoses, and normative developmental trajectories of source-space EEG across spectral organization, connectivity, and signal complexity are established.
An atlas-based ESI pipeline for 30-channel mouse EEG is built and tested against simulated dipoles, but cannot show that a labeled region is the true source in a live animal, and replication across recording systems, sex, age and injection-naive animals is needed.
It is shown that FMR1 gene therapy using delivery routes and vehicles approved for clinical use improves core phenotypes in a mouse model for FXS, and suggests that a dual route may be needed to achieve full brain coverage.
It is demonstrated that FMRP deficiency drives the aberrant translational upregulation of core m6A writers, a causal relationship definitively validated using CGG-corrected isogenic control lines and suggest a promising avenue for m6A-targeted therapies.
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