Gallic Acid, Renal Redox Balance and NRF2-Related Transcript-Level Modulation in a Rat Model of Contrast-Associated Acute Kidney Injury.
GA was associated with biochemical and transcript-level attenuation of acute diatrizoate-induced CA-AKI, mainly through improved renal redox balance and restoration of NRF2-related transcripts, however, functional renoprotection and NRF2 pathway activation were not definitively demonstrated.