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Investigations using various experimental techniques reveal that faulty neuroplasticity develops before the formation of visible protein aggregates, leading to synaptic dysfunction and specific loss of striatal medium spiny neurons in Huntington’s Disease.
Klotho is a promising neurochemical modulator with disease-modifying properties in HD, and its multifunctional protective activities are consistent with important pathological markers of HD, necessitating more preclinical and clinical studies to confirm its translational value.
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