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G. Blanco

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Open access Aug 2026

Stress granules confer an early vulnerability in KRAS-driven pancreatic tumorigenesis.

It is shown that stress granules (SGs) formation is a key adaptive mechanism enabling pancreatic cells to tolerate oncogenic KRAS signaling, and SGs inhibition does not affect pancreatic damage during chronic pancreatitis, supporting its safety for selectively targeting KRAS-mutant cells.

Matías Estarás, Emma Cosialls, Gareth Pollin et al. · 0 citations

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