Background: Severe nutritional Vitamin D deficiency leading to secondary hyperparathyroidism can clinically mimic or mask underlying genetic bone disease. A key challenge is distinguishing whether diffuse skeletal osteosclerosis represents a metabolic reaction to elevated parathyroid hormone (PTH) or a primary defect i...
Tahmina Rahman, T. Tofail, H. Banu et al.· Journal of Association of Cl...· 0 citations
Background: Spontaneous hypoglycemia in non-diabetic adults is an uncommon clinical entity. Endogenous hyperinsulinism caused by a metastatic pancreatic neuroendocrine tumor (malignant insulinoma) represents a rare and critical diagnosis requiring prompt identification and medical stabilization when surgical cure is pr...
Mariam Sultana, Zarin Tasnim Siddiqua, Nur Nahar Begom et al.· Journal of Association of Cl...· 0 citations
Background: Pseudohypoparathyroidism type 1A (PHP1A), now classified within the spectrum of inactivating PTH/PTHrP signaling disorders (iPPSD), is often recognized by the stereotypical Albright hereditary osteodystrophy (AHO) phenotype and parathyroid hormone (PTH) resistance. However, subtle or atypical phenotypic exp...
Abdullah Al Noman Bhuiyan, Nur-E-Haseen Prodhan, Fahmida Akther et al.· Journal of Association of Cl...· 0 citations
Background: Obesity exacerbates metabolic and androgenic abnormalities in polycystic ovary syndrome (PCOS). Although metformin is widely used, its effects on weight and metabolic dysfunction are modest. Semaglutide may offer additional benefit, but evidence in South Asian women with PCOS and obesity is limited.
Objecti...
Md. Rafayel Islam, Md Shahed-Morshed, H. Banu et al.· Journal of Association of Cl...· 0 citations
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