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The net skeletal effect of the immune system is determined by the balance of interacting cell types, cytokine networks, and signaling pathways, and systematic evaluation of combination strategies pairing immunomodulators with anti-resorptive, anabolic agents, or senolytics.
This review systematically summarizes the key features of BMSC senescence, including impaired proliferation and differentiation, epigenetic dysregulation, and the acquisition of a pro-inflammatory senescence-associated secretory phenotype (SASP).
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