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Author

Heyuan Zhao

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Aug 2026

MYC-driven BYSL overexpression promotes hepatocellular carcinoma by suppressing nucleolar stress and inactivating the RPL5/RPL11-MDM2-p53 pathway.

It is revealed that BYSL acts as a pivotal downstream mediator of MYC-regulated ribosome biogenesis and promotes hepatocellular carcinoma progression, suggesting that BYSL may represent a potential therapeutic target for hepatocellular carcinoma.

Heyuan Zhao, Hui-Ying Liu, Xia Liu et al. · 0 citations

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