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EspF-TRIM25-NLRP3 axis is a novel mechanism of mycobacterial pathogenesis that drives hyperinflammation to facilitate bacterial survival and dissemination.
This study highlights the S32 site in the PRRSV GP5 protein as an important factor influencing virus propagation, neutralization, cell tropism and pathogenicity in piglets, and suggests that targeting this residue could lead to the development of more effective PRRSV vaccines.
Transcriptional analysis revealed that compared to neutral conditions, osmotic stress-related genes were significantly upregulated in the ΔliaS strain under acidic conditions, indicating that LiaS modulates acid tolerance through transcriptional regulation.
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