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J. P. Stohn

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Open access Oct 2026

Thyroid hormone excess targeted to oxytocin neurons disrupts oxytocin physiology without impacting behavior.

T3 excess in mouse oxytocin neurons is sufficient to cause significant oxytocin deficits, but insufficient per se to cause detectable abnormalities in behavioral profiles, suggesting that the profound behavioral syndrome previously observed in mice with systemic developmental thyrotoxicosis likely involves other brain...

J. P. Stohn, Zhao-Fei Wu, Arturo Hernandez · 0 citations

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