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Author

Jacob Andresen

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Open access Aug 2026

Aging preserves mTORC1 but attenuates JNK-SMAD2L signaling sensitivity to passive stretch-induced tension development in isolated mouse skeletal muscle.

The findings reveal that mechanosensitive anabolic signaling is differentially affected by aging, and suggests that age-related deficits observed in vivo may, at least in part, arise from systemic influences rather than intrinsic defects adhering to mTORC1 mechanotransduction.

J. B. Kristiansen, Jacob Andresen, Jesper Emil Jakobsgaard et al. · 0 citations

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