Author

K. Choe

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Open access Sep 2026

Autism-risk gene mutations convergently disrupt sexually dimorphic oxytocin circuits to lower social engagement

Autism arises from diverse genetic risk factors, yet how they converge to produce core symptoms and contribute to its sex bias remains unestablished. Oxytocin increases sociability in multiple murine autism models, presenting an opportunity to identify a potentially shared mechanistic basis across etiologies. Here we s...

A. Patwardhan, A. Moslemian, E. Tse et al. · bioRxiv · 1 citation

#small language model Open access Sep 2026

Cntnap2 deletion in mice causes developmentally transient alterations in auditory processing and persistent startle hyperreactivity

Autism spectrum disorder (ASD) is a neurodevelopmental condition often associated with auditory symptoms, including impairments in sound perception and hypersensitivity. These symptoms are thought to be partly due to the delayed development of the auditory brainstem, as evidenced through aberrant auditory brainstem res...

N. Chan, S. D. I. Seheult, A. Warr et al. · bioRxiv · 0 citations

Open access Aug 2026

Intranasal oxytocin modulates the social salience network in adult men with autism

These findings provide first evidence for clinical relevance of the SSN in humans with autism and highlight circuits that may represent promising biomarkers for predicting oxytocin responsiveness.

J. G. RenstroÌm, J. Prinsen, K. Alaerts et al. · medRxiv · 0 citations