Author

Kenneth S. Ramos

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Review Open access Sep 2026

Exploiting DNA damage tolerance for precision oncology.

Unresolved DNA lesions trigger replication stress, forcing cancer cells to hijack DNA damage tolerance (DDT) networks, specifically translesion synthesis (TLS) and template switching, to sustain replication. While DDT prevents lethal fork collapse, error-prone TLS drives mutagenesis, tumor evolution, chemoresistance an...

Audesh Bhat, G. Lahane, R. Pandita et al. · Trends in Cancer · 0 citations