ACP5‐positive macrophages contribute to cerebral oedema and neuroinflammation after traumatic brain injury
Abstract Background Traumatic brain injury (TBI) is a leading cause of death and long‐term disability worldwide. Secondary cerebral oedema, driven by neuroinflammation and blood–brain barrier (BBB) disruption, critically determines poor outcomes, yet effective targeted therapies remain absent. This study aimed to ident...