Molecular simulation reveals how the host thromboinflammatory C-type lectin receptor CLEC2 interacts with the receptor-binding domain of SARS-CoV-2
Recent studies highlight the pivotal role of a type II transmembrane spleen tyrosine kinase-coupled C-type lectin as a novel pattern recognition host-cell receptor (CLEC2) in driving intense proinflammatory responses through platelet hyperactivation and neutrophil extracellular trap (NET) aggregation upon binding wit...