Platelet–NK cell interplay in advanced coronary artery disease is associated with pre-exhausted NK cell dysfunction without upregulation of the immune checkpoint receptor NKG2A
Platelet crosstalk with leukocytes plays a key role in the development of atherosclerosis. However, different immune cells are involved in the disease’s pathology, with Tregs showing protective effects against atherosclerosis, whereas NK cells appear to contribute to both atherogenic and atherothrombotic events. Given...