Interleukin-35 attenuates hepatic lipotoxic stress through AMPK-associated metabolic and antioxidative regulation in NAFLD-like models.
Nonalcoholic fatty liver disease (NAFLD) is characterized by dysregulated hepatic lipid metabolism and oxidative stress under chronic metabolic overload. AMP-activated protein kinase (AMPK) is a central regulator of metabolic and redox homeostasis; however, immune-derived signals that coordinate metabolic adaptation in...