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Miriam Engels

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Open access Aug 2026

Overactive EAAT1 Cl− channels impair GABAergic tonic inhibition in SLC1A3-associated episodic ataxia

It is demonstrated how increased EAAT1 chloride channel activity of the P290R variant can cause hyperexcitability by modifying synaptic GABA concentrations, while impaired EAAT1 glutamate transport leaves glutamatergic synaptic transmission in Slc1a3P290R/+ mice unaffected.

Yulia Kostritskaia, Daniela Bulla-Miely, Miriam Engels et al. · 0 citations

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