Targeting Keap1/Nrf2/Ho-1 Pathway May Explain the Ameliorative Effect of Carnosic Acid against Cyclophosphamide-Induced Cardiotoxicity
Co-treatment with CA effectively attenuates Cyclo-induced cardiotoxicity through its antioxidant, anti-inflammatory, and anti-apoptotic properties, highlighting CA’s therapeutic potential as a natural cardioprotective agent in chemotherapy-associated cardiac injury.