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Open access Jul 2026

Intranasal Exposure of NCM811 Induces Pulmonary Injury by Driving Phenotypic Alveolar Macrophages Reprogramming via Lysosome-Peroxisome Crosstalk

These findings prioritize the lungs for surveillance in NCM811 exposure, identifying the injurious phenotypic shift of alveolar macrophages as a cellular biomarker for early detection and the protease-driven lysosome-peroxisome crosstalk as a key therapeutic target.

Xueyu Zhang, Ze Zhang, Ning Fu et al. · 0 citations

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