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Author

O. Adebiyi

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Open access Aug 2026

Myelin maturation failure and oligodendrocyte precursor loss underlie default mode network disruption in a humanized APP knock-in model of Alzheimer’s disease

Findings identify disruption of myelin–network coupling as a potential mechanism underlying the biphasic evolution of network dysfunction in Alzheimer’s disease and suggest that restoring oligodendroglial function may provide cognitive benefits beyond those of amyloid-targeting strategies.

Esmin Unaran, O. Adebiyi, Medha R. Krishnan et al. · 0 citations

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