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Author

Pei-Hsun Cheng

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Open access Sep 2026

miR-196a reduces mutant Huntingtin aggregates by Rad23b-mediated degradation in Huntington’s disease

Findings demonstrate that critical role of Rad23b in miR-196a-reduced pathological aggregates is demonstrated, and highly suggest downregulating Rad23b or disrupting its interaction with mHTT may offer novel strategies to mitigate mHTT aggregates to delay disease progression.

Chih-Wei Tung, Siew Chin Chan, Yi-Ching Chen et al. · 0 citations
Open access Aug 2026

Wild-type C9orf72 drives proteasomal dysfunction and mutant aggregates via a Stat1-Isg15 axis in Huntington's disease.

A previously unrecognized modulatory role of wt-C9orf72 in regulating mHTT aggregation in experimental HD models is identified and a novel wt-C9orf72-Stat1-Isg15 axis is identified, providing new insights into wt-C9orf72-associated protein homeostasis.

Siew Chin Chan, Chih-Wei Tung, Chih-Yi Chang et al. · 0 citations

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