Real-world PM2.5 exposure triggers early testicular epigenetic responses in the absence of significant changes in measured reproductive endpoints.
BACKGROUND Industrialization has led to widespread air pollution, among which particulate matter ≤ 2.5 μm (PM2.5) represents a significant environmental pollutant associated with various adverse health outcomes. Emerging evidence indicates that the male reproductive system is a sensitive target organ of PM2.5 toxicity;...