Complement-dependent phagocytosis in the clearance of various cellular targets.
It is concluded that complement is necessary for the optimal clearance of apoptotic cells, pathogens, myelin, cellular debris and weak synapses.
2 papers indexed here
We haven’t gathered this author’s papers yet. Follow them and we’ll fetch their work.
Not the right person? Other researchers publish under this name.
It is concluded that complement is necessary for the optimal clearance of apoptotic cells, pathogens, myelin, cellular debris and weak synapses.
C3b-specific nanobodies are developed by generating phage libraries from llamas immunized with purified and surface-bound C3b and Functionally, UNbC3b-1 inhibits complement activity in the alternative pathway (AP), likely by preventing association between substrate C3 and the AP C3 convertase (C3bBb).
We use cookies to run the site and, with your consent, for analytics and to show ads. See our Cookie Policy.