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The MC-restricted St8sia1-GD3 axis functions as a glyco-epigenetic checkpoint driving maladaptive cardiac remodeling, and Targeting this axis represents a translatable immunomodulatory strategy to prevent the progression to chronic heart failure.
Conurrently high TyG and SIRI identifies individuals at significantly increased mortality risk and this simple, threshold-based stratification may serve as a practical tool for cardiovascular risk assessment.
Findings reveal that Lidd selectively targets the PFKFB3-mediated glycolytic-epigenetic axis to suppress PASMC phenotypic transformation and pulmonary vascular remodeling, positioning it as a promising therapeutic candidate for PH.
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