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Sofie De Prijck

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Open access Jul 2026

TNFAIP3/A20 dysfunction drives innate and sterile hyperinflammation

It is suggested that in diseases caused by insufficient TNFAIP3/A20 activity, autoantibodies may reflect a downstream consequence of disease rather than a causative driver, suggesting autoinflammatory rather than autoimmune pathology.

Karel F. A. Van Damme, Pieter Hertens, D. Sichien et al. · Frontiers in Immunology · 0 citations