Author

U. Sure

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Open access Aug 2026

PDCD10-Deficient Tumor Cells Reprogram Angiogenesis Through Paracrine Endothelial Activation and GSC-like Plasticity in GBM

It is demonstrated that PDCD10 loss promotes GBM neo-angiogenesis involving complementary paracrine and GSC-like plasticity mechanisms, highlighting PDCD10 as a potential therapeutic target to suppress neo-angiogenesis in GBM.

Zhong-Rong Chen, Zhen Chen, X. Wan et al. · Cells · 0 citations