D‐Psicose Attenuates Hepatic Steatosis and Inflammatory Injury in HFD/STZ‐Induced T2DM Mice and Is Associated With Alterations in Gut Microbiota, Tryptophan Metabolism, and Hepatic AhR‐Related Signaling
DPS improved metabolic and liver‐related abnormalities in diabetic mice and was accompanied by coordinated changes in intestinal barrier‐related indices, gut microbial composition, tryptophan metabolites, and hepatic AhR/NF‐κB‐related markers, which do not demonstrate a causal gut microbiota–tryptophan–AhR mechanism.