Open access
Jul 2026
Intranasal Exposure of NCM811 Induces Pulmonary Injury by Driving Phenotypic Alveolar Macrophages Reprogramming via Lysosome-Peroxisome Crosstalk
These findings prioritize the lungs for surveillance in NCM811 exposure, identifying the injurious phenotypic shift of alveolar macrophages as a cellular biomarker for early detection and the protease-driven lysosome-peroxisome crosstalk as a key therapeutic target.
Xueyu Zhang, Ze Zhang, Ning Fu et al.
· Environment & Health · 0 citations