IGF2BP3 Orchestrates Metabolic Reprogramming via Stabilizing m6A-Modified PLK1 mRNA to Promote Hepatocellular Carcinoma Progression
Epigenetic dysregulation, particularly N6-methyladenosine (m6A) modification, drives hepatocellular carcinoma (HCC) progression, yet the oncogenic role of the m6A reader IGF2BP3 remains incompletely understood. This study aimed to elucidate the functional contributions and molecular mechanisms of IGF2BP3 in HCC. Multi-...