S100A8/A9 decorated neutrophil extracellular traps contributes to endothelial cell dysfunction via impairing autophagic axis in sepsis lung injury.
Endothelial dysfunction underpins the pathogenesis of sepsis-induced acute lung injury (SI-ALI), wherein neutrophil extracellular traps (NETs) serve as pivotal mediators driving excessive inflammatory responses and progressive tissue deterioration. This study aimed to elucidate the molecular mechanism by which NETs dis...