TL;DR
This review presents a practical, generalizable framework for rehabilitative care and illustrates its application across three diagnoses spanning nerve and muscle disease, including amyotrophic lateral sclerosis, neuralgic amyotrophy, and Duchenne muscular dystrophy.
This thesis addresses isolated cervical dystonia (iCD) assessment and rehabilitation by integrating motor and non-motor symptom management through scalable, evidence-based rehabilitation strategies. Despite advancements in iCD management, such as Botulinum neurotoxin (BoNT) injection, substantial numbers of patients co...
PURPOSE Improved survival from critical illness has produced a growing population of intensive care unit (ICU) survivors with new or worsened physical, cognitive, and mental-health impairment, defined as post-intensive care syndrome (PICS), and frequent distress among families/caregivers (PICS-family, PICS-F). This nar...
Exercise is central to neurological rehabilitation, yet standardized, one-size-fits-all prescriptions frequently fail to reflect the heterogeneity of impairment, disease trajectory, and personal context that characterize conditions such as stroke, Parkinson's disease (PD), multiple sclerosis (MS), spinal cord injury (S...
Background Current neurorehabilitation often relies on static, diagnosis-driven protocols or generic exercise administration, contributing to suboptimal long-term outcomes. There is a clinical need for a cohesive, mechanism-based framework to guide therapeutic reasoning, dosing, and longitudinal adaptation. Methods A s...
BACKGROUND AND OBJECTIVES Patients with late-stage parkinsonism face complex, progressive symptoms requiring specialized interdisciplinary care, yet the unique needs of these patients and family care partners are often inadequately addressed in standard neurology practice. Evidence supporting innovative palliative care...
Background: 5q spinal muscular atrophy (5q SMA) is an autosomal recessive neuromuscular disorder caused by mutations in the survival motor neuron 1 gene (SMN1), leading to deficiency of SMN protein and a variable clinical spectrum influenced by survival motor neuron 2 gene (SMN2) copy number. The advent of disease-modi...
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