Bridging Local and Population Causal Effects: A Proximal Instrumental Variable Approach

Abstract

Instrumental variable (IV) methods address treatment endogeneity, but with non-compliance and heterogeneous treatment effects a binary instrument generally identifies the local average treatment effect (LATE) among compliers rather than the population average treatment effect (ATE). When treatment effects and compliance probabilities are heterogeneous and dependent through latent factors, the ATE need not be identified by IV variation alone. We develop a proximal instrumental variable framework that uses proxies for these factors to adjust for the compliance weighting in LATE and recover the ATE. We show that the ATE is identified through either an outcome bridge capturing latent treatment effect heterogeneity or a compliance bridge representing inverse compliance probabilities, and combining the two yields a doubly robust representation. We derive the efficient influence function under known and unknown instrument propensities. For estimation, we propose regularized kernel minimax bridge estimators, with orthogonalized conditional moments for estimated propensity, and a three-way sequential cross-fitting scheme. We establish projected bridge-risk bounds, and give conditions for asymptotic normality and semiparametric efficiency. The framework provides a proxy-based route from local IV effects to population causal effects under latent treatment effect and compliance heterogeneity.