An agent-based model of a fatal infectious disease with an imperfect immunity on an initially scale-free network that evolves via demographic and epidemic-induced changes and undergoes an epidemic transition between a phase in which the disease eventually dies out and a phase in which it becomes endemic.
ThankGod I. S. Ikpe, Takayuki Hiraoka, N. Fujiwara· 0 citations
While the coupling between traffic dynamics and epidemic spreading on complex networks has been widely studied, existing research has predominantly focused on single-pathogen transmission. In this paper, we investigate the sequential spreading dynamics of two interacting epidemics driven by traffic flow. The model inco...
Xingli Jing, Mao-Bin Hu, Ming Tang et al.· Chaos· 0 citations
In this paper, the complex epidemic behavior of higher-order interactions is investigated by considering heterogeneous hypernetworks with collective contagion mechanisms, and a susceptible-infected-recovered-susceptible epidemic model is established in hypernetworks. First, we propose a mean-field model in heterogeneou...
This work investigates the susceptible-infected-susceptible epidemic model on clustered regular networks using the pair mean-field approach, where higher-order effects are explicitly considered in dynamic analysis and proves the effectiveness of the pair mean-field method for analyzing higher-order network dynamics.
Qing-Chu Wu, Zhao-Yan Wu· Journal of Mathematical Biol...· 0 citations
Network epidemic simulation enables fine-grained understanding of epidemic behavior. However, empirical samples of interaction networks display properties that are challenging to capture with popular synthetic models of networks. Our empirical results show that epidemic spread behavior is sensitive to a form of multi-s...
Omar Eldaghar, M. Mahoney, D. Gleich· PLOS Complex Systems· 0 citations
Extreme epidemic risk is controlled by the right tail of the outbreak-size distribution, but this distribution is generally unknown for non-Markovian spreading on networks. Here we determine this distribution by mapping non-Markovian SIR dynamics to an effective Markovian description. We show that arbitrary infection a...
Ami Taitelbaum, Michael Assaf· 0 citations
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