NtWHY1 negatively regulates tobacco cold tolerance via interfering with flavonoid biosynthesis

Transcriptomic Analysis Reveals the Molecular Mechanisms Underlying Heat-Induced Suppression of Polymethoxyflavone Accumulation in Citrus Leaves

It is suggested that heat stress suppresses PMF accumulation through coordinated repression of PMF biosynthetic genes and their potential regulators and offers potential targets for maintaining citrus nutritional quality under elevated temperatures.

Transcription factor NtELF3 promotes the polyphenol accumulation by targeting NtFLS-1 and NtCHIL-2 genes in tobacco.

It is found that the tobacco transcription factor EARLY FLOWERING 3 (NtELF3), which is highly expressed in tobacco leaves, positively regulates the accumulation of chlorogenic acid, neochlorogenic acid, cryptochlorogenic acid, rutin, scopoletin, and total polyphenols in tobacco middle leaves.

Overexpression of PlPAT1, a GRAS transcription factor from Paeonia lactiflora, enhances salt stress tolerance in transgenic tobacco

It is demonstrated that PlPAT1 functions as a positive regulator of salt stress tolerance, likely through modulating osmotic balance and enhancing reactive oxygen species scavenging capacity.

Functional identification of the bHLH transcription factor MdSAT1 in response to Zn stress in apple

Functional characterization of the MdFLZ2 gene in drought and salt stress tolerance in apple.

A genome-wide characterization of the FLZ gene family in apple and functionally characterized MdFLZ2 suggested MdFLZ2 as a positive regulator of drought and salt tolerance and highlighted its potential to serve as a genetic resource for abiotic stress improvement.

MdXERICO negatively regulates salt tolerance in apple through ubiquitination and degradation of MdJAZ10.

A novel MdXERICO-MdJAZ10 regulatory module is uncovered, in which MdXERICO negatively modulates apple salt tolerance by targeting MdJAZ10 for ubiquitin-dependent degradation, thereby suppressing the expression of ion-transport-related genes and disrupting sodium-potassium balance.