TL;DR
Different patterns of spontaneous brain activity associated with cognitive impairment in SCA3 are revealed, providing novel imaging evidence for the pathophysiological mechanisms underlying cognitive decline in these patients.
Chronic kratom consumption may alter brain activity, especially in areas linked to the DMN, cognition, and reward processing, and enhanced FC between salience and cerebellar networks may reflect compensatory mechanisms or adaptations to kratom's effects.
ABSTRACT Background Cognitive impairment is a prevalent and debilitating feature in multiple sclerosis (MS), yet its pathophysiology remains incompletely understood. Dysfunction of the neurovascular unit (NVU) and impaired neurovascular coupling (NVC) may be related to cognitive performance in MS, but this pathway rema...
Spatial correspondences between T1DM-related brain functional alterations and normative distributions of multiple neurotransmitter receptors/transporters are demonstrated.
Modulating FC between these hub brain regions (R-DLPFC and PCC) may provide new therapeutic strategies for cognitive recovery after DNICH and abnormal coordination of intra-network and inter-network FC of ECN and DMN may represent a shared neural mechanism of cognitive impairment after DNICH.
The authors' multimodal findings suggest subregion- and laterality-specific alterations in the SN of patients with TLE, with patterns differing between RTLE and LTLE.
Previous studies have demonstrated widespread regional abnormalities in blood oxygen level-dependent (BOLD) amplitude of low frequency fluctuation (ALFF) in psychotic disorders. How regional ALFF is related to functional connectivity and behavior in psychotic illness, however, is poorly understood. Here we examined v...
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