Additive effects of heat and volatile organic compounds co-exposure on metabolic syndrome: evidence from petrochemical workers in China

Mixed Exposure to Underground Air Pollutants and Metabolic Syndrome in Coal Miners: A Cross-Sectional Study Integrating Multi-Pollutant Models and Urinary Metabolomics

Logistic regression analysis revealed that exposure to coal dust, carbon monoxide, carbon dioxide, carbon dioxide, and nitrogen dioxide was associated with an increased risk of MetS, and BKMR and Qgcomp models consistently indicated a significant positive association between mixed pollutant exposure and MetS risk.

Occupational Heat Exposure and Kidney Stone Risk in High-Risk Workers: A Systematic Review

Background: Kidney stone disease (KSD) or Nephrolithiasis is an escalating occupational health challenge in high-heat environments, including military, mining, agricultural, and construction sites. Existing evidence remains fragmented regarding how occupational heat stress interacts with lifestyle determinants and meta...

Occupational heat exposure, nighttime warming, and eGFR loss in petrochemical workers: a retrospective cohort study with metabolomic evidence.

Under the context of climate warming, whether rising nighttime temperatures modify heat-related kidney risks among industrial workers remains unclear. This retrospective cohort study included 2,148 petrochemical workers in southeastern China followed from 2013 to 2022. Occupational heat exposure was assessed using the...

The association between carbon monoxide and cardiovascular-kidney-metabolic syndrome in the Chinese populations: China Health and Retirement Longitudinal Study.

[Association between air pollution exposure and metabolic dysfunction-associated steatotic liver disease in adults in China].

Exposures to PM2.5, PM10, NO2, and warm-season O3, as well as their mixture, were associated with increased risk for MASLD and MASLD with significant liver fibrosis in adults in China.

Targetable Preventive Metabolites: Revealing the Pathways from Air Pollution Mixtures to Hypertension Through Integrated Exposure-Metabolomics

C6-carnitine and other preventable metabolites are important biological mediators of air pollution mixture-induced hypertension, partially illuminating the molecular link between environmental exposure and cardiovascular metabolic disorders.