TL;DR
It is demonstrated that NtRhoGAP1, a RhoGAP protein from Nicotiana tabacum, confers resistance to ChiVMV by potentiating salicylic acid (SA) defense signaling.
This study reveals a previously unknown role of COBRA-like proteins in PMAD and provides insight into how a plant viral MP sabotages PMAD through perturbing COBL3-PDLP5 interaction to facilitate virus spread through PD.
A mechanism by which the small GTPase RHO1 regulates ROS compartmentalization to affect the plant response to different pathogen infections is demonstrated, indicating the diversity of responses of a plant gene to different biotic factors.
It is demonstrated that the TaLYK5-TaDSK2a module functions as a molecular switch that dynamically regulates the trade-off between plant immunity and growth.
It is proposed that this endogenous regulation balances the intensity of R8-mediated immunity, likely reducing selection pressure on the pathogen population and prolonging resistance durability, providing a conceptual framework for durable resistance breeding.
It is established that the C4–SlTFT2 interaction is indispensable for TYLCV‐mediated immunosuppression, and C4 promotes successful TYLCV infection by reducing the nuclear accumulation of SlTFT2 through this interaction, revealing a novel mechanism by which the virus subverts host defence by targeting a 14‐3‐3 protein.
Abstract Plant responses to pathogens often rely on receptor-like cytoplasmic kinases (RLCKs) that mediate signaling through interactions with receptor kinases and downstream components. Here, we studied the tomato RLCK, TPK1b Related Protein Kinase (TPK09), and demonstrate its function in integrating defense with ligh...
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