TL;DR
The stable expression of Nrf2, p53, and CALB1 suggests that the favorable clinical performance of MTA may depend not only on biocompatibility but also on its ability to preserve intracellular equilibrium during early tissue contact.
Background and Objectives: Oxidative stress-induced mitochondrial dysfunction contributes to granulosa cell apoptosis and ovarian dysfunction. This study investigated the therapeutic potential of human Wharton’s jelly-derived mesenchymal stem cell-derived mitochondria (hWJ-MSCs-mito) in an H2O2-induced oxidative stress...
Rotenone is a well-defined inhibitor of Mitochondrial Complex I, which induces oxidative stress and apoptosis in various cell types. However, its cytotoxicity and molecular mechanisms in human umbilical vein endothelial cells (HUVECs) remain unexplored. Therefore, the purpose of this study was to examine the effects of...
It is demonstrated that IL-1β induces mitochondrial dysfunction in dermal fibroblasts and establish a reliable inflammatory model for investigating mitochondrial mechanisms involved in skin aging, chronic wound healing, and other inflammation-related skin disorders.
Background: Oxidative stress, neuroinflammation, impaired autophagy, and mitochondrial dysfunction are major contributors to ageing and neurodegenerative diseases. This study investigated whether AgeViva could counteract these processes by modulating redox balance, inflammation, autophagy, and mitochondrial function. M...
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