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This work identifies Treg destabilization as a staged and branching differentiation process rather than an abrupt loss of lineage identity, and establishes a framework for Treg destabilization during atherosclerosis.
To better understand the factors driving Treg to exTreg conversion, transcriptomes of Treg subsets expressing varied levels of CD25 and FOXP3 are analyzed in human peripheral blood CD4+ T cells.
Atherosclerosis is an immune-mediated disease in which regulatory T cells (Tregs) can lose lineage stability and convert into pro-inflammatory effector states (“exTregs”) that may accelerate plaque progression. However, the diversity, lineage relationships, and pathogenic roles of exTreg subsets in cardiovascular...
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