Author

M. Liebau

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#protein folding Open access Oct 2026

Structural modelling predicts that fibrocystin destabilisation is a pathogenic cause of ARPKD

Destabilisation of fibrocystin may be the predominant pathogenic mechanism in ARPKD survivors, and highlight fibrocystin stabilisation as a therapeutic strategy.

Travis A. K. Bannell, Naveen Mathew, K. Burgmaier et al. · bioRxiv · 0 citations

#protein folding Open access Aug 2026

The fibrocystin C-terminal domain inhibits Src/STAT3 signal induced cystogenesis of kidney epithelial cells.

FPCct appears to act like a physiological suppressor of cystogenic signaling, as found in healthy kidney epithelia, that is essential for maintaining epithelial homeostasis.

F. Hassan, Susanne T. Hahnenstein, A. Kraus et al. · American Journal of Physiolo... · 0 citations