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Results indicate a possible link between transcriptional remodeling, RNA editing, and functional polarization of HCC-induced neutrophils, and provide an exploratory framework for investigating ADAR-regulated RNA editing in tumor-immune communication.
The results show that CDSs are rarely predefined in the genome, and the differential roles of predefined versus variable CDS architectures may reflect how natural selection balances molecular stability and functional innovation.
The findings conservatively indicate that Smart-Seq may not be good at analyzing molecular diversity and that the results need to be interpreted with caution.
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