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The genetic architecture of liver triglyceride content is refined, linking polygenic susceptibility to downstream metabolic and inflammatory pathways and providing insights into the genetic basis of steatotic liver disease.
Findings indicate that clinical consequences are determined by the underlying biological mechanism rather than the degree of cholesterol lowering, and genetic testing may be considered in people with metabolic dysfunction-associated steatotic liver disease (MASLD) and hypocholesterolemia to clarify the mechanism, guide...
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