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ADAR2-mediated A-to-I editing confers an oncogenic role on miR-376a-3p by shifting its target from KPNA4 to HOXD10, thereby facilitating PTC tumorigenesis through enhanced glycolysis.
It is demonstrated that zeylenone suppresses proliferation and promotes apoptosis in TPC-1 cells primarily through suppression of the TIMP-1-mediated FAK/PI3K/AKT signaling pathway, highlighting its potential as a therapeutic candidate for thyroid carcinoma.
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